Posted by Not Bob on January 3, 2003, at 23:01:13
In reply to Re: About Dreaming--from my therapist » Not Bob, posted by beardedLADY on January 3, 2003, at 16:21:19
Hey Bearded...
These are quotes and links from Google.
I'm not just making it up, really.
EEG (Electroencephalogram)
Q: Sleep EEGs! What would they show?
A: Studies have shown that individuals with either unipolar or bipolar depression have sleep patterns different from those of people who do not have mood disorders. The rapid-eye-movement (REM) phase of sleep associated with dreaming occurs earlier in people with mood disorders. People with mood disorders also have more eye movements during REM sleep, less deep or slow-wave sleep, and more problems staying asleep.
Still, this pattern is most pronounced in people who are severely ill; it's not a reliable measure of less severe depression.
http://www.sistahspace.com/WeepingWillows/getdown.html
MAOIs significantly reduces REM (rapid eye movement),
sleep density, REM time and the REM percentage of
total sleep time in patients with major
depression.a. Indeed, all drugs that suppress REM have an antidepressant effect (Vogel et al.,
1990) suggesting that at least some of the antidepressant action of drugs might be due to the
suppression of REM, although the presence of several antidepressants that do not suppress REM
precludes this as the sole mechanism by which antidepressants work
The possibility that coexisting depression, which is known to affect REM sleep, accounted for this divergence of findings was not supported by studies comparing groups of patients with and without major depression in addition to PTSD.55,63 Patients with PTSD had both higher and lower values for REM latency than patients with major depression.
http://www.inchem.org/documents/pims/pharm/pimg025.htm
http://my.webmd.com/content/article/16/1663_51686?
There is a great deal of literature that reviews which aspects of sleep are most associated with depression, when measured by conventional EEG techniques. These include reduced REM latency, i.e. the time between sleep onset to the first appearance of REM (Reynolds & Kupfer, 1987), less short wave sleep, particularly in the first NREM/REM sleep cycle (Kupfer & Reynolds, 1992) and lower production of delta waves in NREM sleep (Kupfer, et al., 1990).
http://www.solent.ac.uk/socsci/agm/homepage.htm
http://www.parkinson.org/sleepdisturbances2.htm
http://rjews.net/v_rotenberg/sleep_in_depression.html
Polysomnographic recordings of depressed patients often reveal reduced slow-wave sleep, an early onset of the first episode of REM sleep, and increased phasic REM sleep.9 A deficit in serotonergic neurotransmission, a relative increase in pontine cholinergic activity, and, perhaps, an excess of nor-adrenergic and corticotropin-releasing–hormone activity have been implicated in the pathogenesis of severe depression.9,10
poster:Not Bob
thread:1982
URL: http://www.dr-bob.org/babble/psycho/20021230/msgs/2097.html